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Clarifying the Complex World of Nutrition Science

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Stop Digestive Discomfort Naturally Without Dangerous Drugs!

  • How to Combat Indigestion, Heartburn, Gas, Bloating & Stomach Pain
  • Why Typical Indigestion Medications are Harmful, Making Matters Worse
  • What You Can Do to Improve Digestion for Long Term Health
  • When Poor Digestion Might Cause Numerous Health Problems

The human body is amazingly resilient and can take a lot of abuse. Eating a Big Mac and fries for lunch, pepperoni pizza for dinner, and a pint of ice cream for dessert won't kill you in the short term. But a poor diet laden with saturated fat and little or no fiber can certainly wreak havoc on your digestive system and overall health. Unfortunately, there are plenty of diseases associated with poor nutrition and digestion that take years to rear their ugly head.

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It's estimated that some form of digestive disorder affects more than 100 million Americans.1 For some people, it's a temporary case of dyspepsia, or indigestion, that may cause a sleepless night or a missed day of work. For others, dyspepsia turns into a chronic state of discomfort and pain that can eventually result in a serious illness.

The foundation of good health & the root cause of disease

Nutrition, digestion, absorption, and bacterial balance all play significant roles in the health of your gastrointestinal tract—and in your overall health. Efficient absorption of the nutrients in the food you eat is at the foundation of good health. According to Deepak Chopra, M.D., digestion allows the nutrients you ingest to be broken down and "then combined again in such a way as to re-create every cell, every organ, every tissue of the human body."2 

Conversely, many health practitioners believe that poor digestion is the root cause of most disease. Although it can take years for the consequences of poor digestion to manifest in serious illness, individuals may complain in the interim of a variety of health complaints including joint pain, headache, fatigue, indigestion, heartburn, or stomach problems.3

What is dyspepsia?

Dyspepsia is a complex set of symptoms, rather than an indication of a specific disease. It is the medical term used for indigestion … and indigestion is the term used to describe pain and discomfort in the upper abdomen or chest that usually develops after a meal. It may also be accompanied by nausea, abdominal bloating, belching, or vomiting.

Dyspepsia may be caused by disease or Helicobacter pylori (H. pylori) infection. It can also be the result of eating too much or too fast—especially when you're feeling stressed—and high-fat foods. Smoking, drinking too much alcohol, using medications that irritate the stomach lining (such as anti-inflammatory drugs), fatigue, and chronic stress can also cause indigestion, or make it worse. Additionally, exercising with a full stomach may cause indigestion so try to plan your workout before a meal or at least an hour afterward.

Some people have persistent indigestion that is unrelated to any of these factors. This type of indigestion—called functional or non-ulcer dyspepsia—may be caused by a problem in the muscular squeezing action of the stomach (motility), or by low stomach acid or low bile flow.

Addison's disease, asthma, celiac disease, chronic auto-immune disorders, depression, eczema, gallstones, gastric cancer, gastritis, hepatitis, osteoporosis, psoriasis, acne roseola, colitis, hair loss, and rheumatoid arthritis are just some of the disorders that have been associated with poor stomach acid output.4

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References

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  2. Chopra, Deepak, M.D. Perfect Digestion: The key to balanced living. New York: Harmony Books, 1995. 

  3. Lukaczer, Dan, N.D. "Gastrointestinal Health and Disease."Nutrition Science News, December, 1996.

  4. Alternative Medicine Review 1997; 2(2):116-127. 

  5. Shmuely H, Obure S, Passaro DJ, Abuksis G, Yahav J, Fraser G, et al. Association between dyspeptic symptoms and Helicobacter pylori infection in Nakuru, Kenya. Emerg Infect Dis [serial online] 2003 Sept [date cited]. http://www.cdc.gov/ncidod/EID/vol9no9/02-0374.htm

  6. Haruma K, Kamada T, Kawaguchi H, Okamoto S, Yoshihara M, Sumii K, Inoue M, Kishimoto S, Kajiyama G, Miyoshi A. Effect of age and Helicobacter pylori infection on gastric acid secretion. J Gastroenterol Hepatol. 2000 Mar;15(3):277-83. 

  7. Krasinski Sd, Russell Rm, et al. Fundic atrophic gastritis in an elderly population. Effect on hemoglobin and several serum nutritional indicators. J Am Geriatr Soc. 1986 Nov;34(11):800-6. 

  8. Grossman MI, Kirsner JB, Gillespie IE. Basal and histalog-stimulated gastric secretion in control subjects and in patients with peptic ulcer or gastric cancer. Gastroenterology 1963;45:15-26. 

  9. S.M. Wildi et al. "The influence of rapid food intake on postprandial reflux: Studies in health volunteers." Am J Gastroenterol, Sept. 2004. Vol.99 No.9

  10. Bupa-the personal health service:
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  12. Noto T, Nagasaki M, Yomota E, Endo T. Gastric mucosal functioning following withdrawal of omeprazole in rats.Arzneimittelforschung. 1998 Jan;48(1):70-3.

  13. Havu N, Mattsson H, Ekman L, Carlsson E. Enterochromaffin-like cell carcinoids in the rat gastric mucosa following long-term administration of ranitidine. Digestion. 1990;45(4):189-95.

  14. Delle Fave G, Helander H, Holt S, et al. Acid suppression and gastric mucosal biology. Dig Dis Sci. 1994 Sep;39(9):1843-52.

  15. Balch, James, M.D., Walker, Morton, M.D. Heartburn and What To Do About It. Avery Publishing Group, Garden City Park, New York, 1998. pp12-14 

  16. National Digestive Disease Education and information Clearinghouse. Cirrhosis of the liver. DD Clearinghouse Fact Sheet. Public Health Service, NIH: U.S. Dept of Health and Human Services, 2989. 

  17.  "When it's not an ulcer." Consumer Reports, August 1995, p. 552.

  18. EMedicine consumer healthhttp://www.emedicinehealth.com/articles/43286-6.asp

  19. http://www.Medicinenet.comhttp://www.medicinenet.com/omeprazole/article.htm

  20. Swidey, Neil. "How the Drug Companies Deceive You: The Inside Story of Nexium." Boston Globe November 17, 2002. 

  21. Jordan, Karin G., M.D. "The Artichoke, More Than Just a Gourmet Food." Life Extension Magazine, July 1999. 

  22. "Artichoke Extract: Improves Digestion, Liver Function, and Cholesterol Levels." Natural Medicine Journal, Aug/Sept. Vol. 1, No.7, 1998. 

  23. Kirchoff R, et al. Increase in choleresis by means of artichoke extract. Results of a randomized placebo-controlled double-blind study. Phytomedicine 1;107, 1994. 

  24. Walker AF, Middleton RW, Petrowicz O. Artichoke leaf extract reduces symptoms of irritable bowel syndrome in a post-marketing surveillance study. Phytotherapy Research, 2001. Vol. 15: 58-61.

  25. Petrowicz O, Gebhardt R, Donner M, Schwandt P, Kraft K: effects of artichoke leaf extract on lipoprotein metabolism in vitro and in vivo.Atherosclerosis 129: 147, 1997. 

  26. Gebhardt R. Antioxidative and protective properties of extract from leaves of the artichoke (Cynara scolymnus L.) against hydro-peroxide-induced oxidative stress incultured rat hepatocytes. Toxicol Appl Pharmacol 144:279-286, 1997.

  27. Hartwell JL. Plants used against cancer. Lloydia 1967;30/4:379-436. 

  28. Huwez FU, Thirlwell D, Cockayne A, Ala'Aldeen DA. Mastic gum kills Helicobacter pylori. NEJM 1998 Dec 24;339(26):1946.

  29. Jones RG, Trowbridge DB, Go MF. Helicobacter pylori infection in peptic ulcer disease and gastric malignancy. Front Biosci 2001 Dec 1;6:E213-26. Review. 

  30. Huwez FU, Thirlwell D, Cockayne A, Ala'Aldeen DA. Mastic gum kills Helicobacter pylori. NEJM 1998 Dec 24;339(26):1946. 

  31. Huwez FU, Al-Habbal MJ. Mastic in treatment of benign gastric ulcers. Gastroenterol Jpn 1986 Jun;21(3):273-4.

  32. Al-Said MS, Ageel AM, Parmar NS, Tariq M. Evaluation of mastic, a crude drug obtained from Pistacia lentiscus for gastric and duodenal anti-ulcer activity. J Ethnopharmacol 1986;15:271-8. 

  33. Al-Habbal MJ, Al-Habbal Z, Huwez FU. A double-blind controlled clinical trial of mastic and placebo in the treatment of duodenal ulcer.J Clin Exp Pharm Physiol 1984;11:541-4. 

  34. Afzal M, Al-Hadidi D, Menon M, Pesek J, Dhami MS. Ginger: an ethnomedical, chemical and pharmacological review. Drug Metabol Drug Interact. 2001;18(3-4):159-90.

  35. Mahady GB, Pendland SL, Yun GS, Lu ZZ, Stoia A. Ginger (Zingiber officinale Roscoe) and the gingerols inhibit the growth of Cag A+ strains of Helicobacter pyloriAnticancer Res. 2003 Sep-Oct;23(5A):3699-702.

  36. al-Yahya MA, Rafatullah S, Mossa JS, et al. Gastroprotective activity of ginger zingiber officinale rosc., in albino rats. Am J Chin Med. 1989;17(1-2):51-6.

  37. Gonlachanvit S, Chen YH, Hasler WL, Sun WM, Owyang C. Ginger reduces hyperglycemia-evoked gastric dysrhythmias in healthy humans: possible role of endogenous prostaglandins. J Pharmacol Exp Ther. 2003 Dec;307(3):1098-103. Epub 2003 Oct 08.

  38. Hughes VL, Hillier SL. Microbiologic characteristics of Lactobacillus products used for colonization of the vagina. Obstet Gynecol. 1990 Feb;75(2):244-8.

  39. La Rosa M, Bottaro G, Gulino N, Gambuzza F, Di Forti F, Ini G, Tornambe E. [Prevention of antibiotic-associated diarrhea with Lactobacillus sporogens and fructo-oligosaccharides in children. A multicentric double-blind vs placebo study][Article in Italian] Minerva Pediatr. 2003 Oct;55(5):447-52.

  40. Mohan JC, Arora R, Khalilullah M. Preliminary observations on effect of lactobacillus sporogenes on serum lipid levels in hypercholesterolemic patients. Indian J Med Res. 1990 Dec;92:431-2.

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  42. Smirnov VV, Reznik SR, V'iunitskaia VA, et al. The effect of the complex probiotic sporolact on the intestinal microbiocenosis of warm-blooded animals. Mikrobiol Z 1995;57:42-49. [Article in Russian]

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